Porcine gastric ulcer.

Research output: Contribution to journalArticle

26 Citations (Scopus)

Abstract

Ulceration of the pars esophagea in swine develops from a complex interaction of dietary particle size, gastric fluidity, dietary carbohydrate content, and presence of certain species of commensal gastric organisms capable of fermenting dietary carbohydrates. Unlike in humans, the significance of the role of Helicobacter sp. in development of porcine gastric ulcers is yet undefined. Management practices that limit the incidence and severity of gastric ulceration without interfering with growth performance appear to be the best option for control.

Original languageEnglish (US)
Pages (from-to)163-174
Number of pages12
JournalThe Veterinary clinics of North America. Food animal practice
Volume16
Issue number1
DOIs
StatePublished - Mar 2000

Fingerprint

stomach ulcers
Stomach Ulcer
Dietary Carbohydrates
Stomach
stomach
dietary carbohydrate
Swine
swine
Helicobacter
Practice Management
carbohydrate content
Particle Size
particle size
growth performance
incidence
Incidence
organisms
Growth

ASJC Scopus subject areas

  • Food Animals

Cite this

Porcine gastric ulcer. / Doster, Alan R.

In: The Veterinary clinics of North America. Food animal practice, Vol. 16, No. 1, 03.2000, p. 163-174.

Research output: Contribution to journalArticle

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